Emma E. Moore - Seattle WA, US Harald S. Haugen - Seattle WA, US
International Classification:
A61K 38/20 A61K 39/395 A61P 17/06
US Classification:
424 852, 4241581
Abstract:
IL-17B is known to stimulate the proliferation of chondrocytes, bone, and is highly expressed in nervous tissue, resulting in repair of diseased tissue. When IL-17B is absent a marked negative effect on wound healing is noted. The present invention comprises providing IL-17B, by topical, parental, or other administration means, in order to accelerate the wound healing process. The present invention further encompasses a pharmaceutical composition and formulations thereof that utilize IL-17B, either alone or in combination with other cytokines or growth factors known to aid wound healing. The invention also contemplates methods of treating wounds in patients using this pharmaceutical composition.
Emma E. Moore - Seattle WA, US Harald S. Haugen - Seattle WA, US
International Classification:
A61K 38/20 A61P 17/02
US Classification:
424 852
Abstract:
IL-17B is known to stimulate the proliferation of chondrocytes, bone, and is highly expressed in nervous tissue, resulting in repair of diseased tissue. When IL-17B is absent a marked negative effect on wound healing is noted. The present invention comprises providing IL-17B, by topical, parental, or other administration means, in order to accelerate the wound healing process. The present invention further encompasses a pharmaceutical composition and formulations thereof that utilize IL-17B, either alone or in combination with other cytokines or growth factors known to aid wound healing. The invention also contemplates methods of treating wounds in patients using this pharmaceutical composition.
Emma Moore - Seattle WA, US Harald Haugen - Seattle WA, US
International Classification:
A61K 38/20
US Classification:
424085200, 424085100, 514012000, 424445000
Abstract:
IL-17B is known to stimulate the proliferation of chondrocytes, bone, and is highly expressed in nervous tissue, resulting in repair of diseased tissue. When IL-17B is absent a marked negative effect on wound healing is noted. The present invention comprises providing IL-17B, by topical, parental, or other administration means, in order to accelerate the wound healing process. The present invention further encompasses a pharmaceutical composition and formulations thereof that utilize IL-17B, either alone or in combination with other cytokines or growth factors known to aid wound healing. The invention also contemplates methods of treating wounds in patients using this pharmaceutical composition.
International Clinical Research Center, Dept of Global Health, Univ Washington since Oct 2008
Specimen Repository Manager
ZymoGenetics Oct 1997 - Feb 2008
Scientist
Bristol-Myers Squibb Apr 1990 - Oct 1997
Associate Scientist
Education:
University of Alaska Fairbanks 1982 - 1986
M.S., BiologyThesis on Arctic wolves
Skills:
Laboratory Clinical Trials Cell Biology Elisa Pharmaceutical Research Clinical Research Biotechnology Pcr Immunology Life Sciences Cell Culture Public Health Drug Discovery Molecular Biology Lifesciences Oncology Drug Development Clinical Development Microbiology Research Biochemistry Pharmaceutical Industry Technology Transfer Glp Protein Chemistry Biopharmaceuticals
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